As compared to SARS-CoV, SARS-CoV-2 appears to have a functional advantage regarding the ectodomain of the S protein and its affinity to ACE2 receptors on host cells.It is estimated that SARS-CoV ...

Recently, some researchers have shown that SARS-CoV-2 infection can be inhibited by a recombinant soluble hACE2 receptor, perhaps by direct competition for the host ACE2 for the viral spike protein.

A SARS-CoV-2 infection begins when the virus’s spike proteins bind to ACE2 cell surface receptors, enabling the virus to infect human cells.

SARS-CoV-2 infection is shown to be codependent on heparan sulfate and ACE2, suggesting new approaches, including repurposed heparin, to treating COVID-19 ...

Despite years of global investigation, the WHO confirms COVID-19’s true origin remains unknown due to limited data, with ...

They then exposed both cell types to SARS-CoV-2. Surprisingly, a lack of ACE2 receptors decreased SARS-CoV-2 infection in the Caco2 control cells but had no effect on infection in the Jurkat cells.

The new COVID-19 variants NB.1.8.1 and XFG are rapidly spreading and competing for dominance across the globe.

"These viruses are so closely related to MERS, so we have to be concerned if they ever infect humans," said virologist ...

Arthur JM, Forrest JC, Boehme KW, et al. Development of ACE2 autoantibodies after SARS-CoV-2 infection. PLoS One 2021;16(9):e0257016-e0257016. Crossref. PubMed. Web of Science. Google Scholar. 2.

SARS-CoV-2 infection in children and adolescents increases risk for cardiovascular disease, compared with uninfected individuals.